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Scientific paper - Case study
Inflammation in prostatic hyperplasia and carcinoma-basic scientific approach
Frontiers in Oncology, 7 (2017); 77. https://doi.org/10.3389/fonc.2017.00077
Krušlin, Božo; Tomas, Davor; Džombeta, Tihana; Milković-Periša, Marija; Ulamec, Monika

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Krušlin, B., Tomas, D., Džombeta, T., Milković Periša, M. & Ulamec, M. (2017). Inflammation in prostatic hyperplasia and carcinoma-basic scientific approach. Frontiers in Oncology, 7., 77. doi: 10.3389/fonc.2017.00077

Krušlin, Božo, et al. "Inflammation in prostatic hyperplasia and carcinoma-basic scientific approach." Frontiers in Oncology, vol. 7, 2017, p. 77. https://doi.org/10.3389/fonc.2017.00077

Krušlin, Božo, Davor Tomas, Tihana Džombeta, Marija Milković Periša and Monika Ulamec. "Inflammation in prostatic hyperplasia and carcinoma-basic scientific approach." Frontiers in Oncology 7 (2017): 77. https://doi.org/10.3389/fonc.2017.00077

Krušlin, B., et al. (2017) 'Inflammation in prostatic hyperplasia and carcinoma-basic scientific approach', Frontiers in Oncology, 7, p. 77. doi: 10.3389/fonc.2017.00077

Krušlin B, Tomas D, Džombeta T, Milković Periša M, Ulamec M. Inflammation in prostatic hyperplasia and carcinoma-basic scientific approach. Frontiers in Oncology [Internet]. 2017 April 25 [cited 2024 May 03];7:77. doi: 10.3389/fonc.2017.00077

B. Krušlin, D. Tomas, T. Džombeta, M. Milković Periša and M. Ulamec, "Inflammation in prostatic hyperplasia and carcinoma-basic scientific approach", Frontiers in Oncology, vol. 7, p. 77, April 2017. [Online]. Available at: https://urn.nsk.hr/urn:nbn:hr:105:485663. [Accessed: 03 May 2024]

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Metadata
Title (english)Inflammation in prostatic hyperplasia and carcinoma-basic scientific approach
AuthorBožo Krušlin
AuthorDavor Tomas
AuthorTihana Džombeta
AuthorMarija Milković-Periša
AuthorMonika Ulamec
Author's institutionUniversity of Zagreb
School of Medicine
Scientific / art field,
discipline and subdiscipline		BIOMEDICINE AND HEALTHCARE
Clinical Medical Sciences
Abstract (english)
Chronic inflammation is associated with both benign conditions and cancer. Likewise, inflammatory cells are quite common in benign prostatic hyperplasia (BPH) and prostatic tissue harboring cancer. Triggers that activate inflammatory pathways in the prostate remain a subject of argument and are likely to be multifactorial, some of these being bacterial antigens, different chemical irritations, and metabolic disorders. Acute and chronic inflammation in prostate leads to accumulation of immunocompetent cells, mainly T lymphocytes and macrophages, but also neutrophils, eosinophils, and mast cells, depending on the type of offending agent. Inflammatory processes activate hyperproliferative programs resulting in nodules seen in BPH, but are also important in creating suitable microenvironment for cancer growth and progression. Inflammatory cells have mostly been shown to have a protumoral effect such as tumor-associated macrophages, but some cell types such as mast cells have antitumoral effects. This review outlines the recent findings and theories supporting the role of inflammatory responses as drivers of both benign and malignant epithelial processes in the prostate gland.
Languageenglish
Publication typeScientific paper - Case study
Publication statusPublished
Peer reviewPeer review
Publication versionPublished version
Journal titleFrontiers in Oncology
Numberingvol. 7, p. 77
p-ISSN2234-943X
DOIhttps://doi.org/10.3389/fonc.2017.00077
URN:NBNurn:nbn:hr:105:485663
Publication2017-04-25
Document URLhttps://doi.org/10.3389/fonc.2017.00077
Type of resourceText
Access conditionsOpen access
Terms of useLicence In copyright icon
Created on2024-04-09 05:47:02