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Iron overload in aging Bmp6 ‑/‑ mice induces exocrine pancreatic injury and fibrosis due to acinar cell loss
International Journal of Molecular Medicine, 47 (2021), 4; 60. https://doi.org/10.3892/ijmm.2021.4893

Pauk, Martina; Kufner, Vera; Rumenović, Viktorija; Dumić‑Čule, Ivo; Farkaš, Vladimir; Milošević, Milan; Bordukalo‑Nikšić, Tatjana; Vukičević, Slobodan

Citirajte ovaj rad

Pauk, M., Kufner, V., Rumenović, V., Dumić‑Čule, I., Farkaš, V., Milošević, M. ... Vukičević, S. (2021). Iron overload in aging Bmp6 ‑/‑ mice induces exocrine pancreatic injury and fibrosis due to acinar cell loss. International Journal of Molecular Medicine, 47. (4). doi: 10.3892/ijmm.2021.4893

Pauk, Martina, et al. "Iron overload in aging Bmp6 ‑/‑ mice induces exocrine pancreatic injury and fibrosis due to acinar cell loss." International Journal of Molecular Medicine, vol. 47, br. 4, 2021. https://doi.org/10.3892/ijmm.2021.4893

Pauk, Martina, Vera Kufner, Viktorija Rumenović, Ivo Dumić‑Čule, Vladimir Farkaš, Milan Milošević, Tatjana Bordukalo‑Nikšić i Slobodan Vukičević. "Iron overload in aging Bmp6 ‑/‑ mice induces exocrine pancreatic injury and fibrosis due to acinar cell loss." International Journal of Molecular Medicine 47, br. 4 (2021). https://doi.org/10.3892/ijmm.2021.4893

Pauk, M., et al. (2021) 'Iron overload in aging Bmp6 ‑/‑ mice induces exocrine pancreatic injury and fibrosis due to acinar cell loss', International Journal of Molecular Medicine, 47(4). doi: 10.3892/ijmm.2021.4893

Pauk M, Kufner V, Rumenović V, Dumić‑Čule I, Farkaš V, Milošević M, i sur.. Iron overload in aging Bmp6 ‑/‑ mice induces exocrine pancreatic injury and fibrosis due to acinar cell loss. International Journal of Molecular Medicine [Internet]. 04.2021. [pristupljeno 20.01.2022.];47(4). doi: 10.3892/ijmm.2021.4893

M. Pauk, et al., "Iron overload in aging Bmp6 ‑/‑ mice induces exocrine pancreatic injury and fibrosis due to acinar cell loss", International Journal of Molecular Medicine, vol. 47, br. 4, Travanj 2021. [Online]. Dostupno na: https://urn.nsk.hr/urn:nbn:hr:105:037867. [Citirano: 20.01.2022.]

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