prikaz prve stranice dokumenta The Impact of Modern Anti-Diabetic Treatment on Endothelial Progenitor Cells
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The Impact of Modern Anti-Diabetic Treatment on Endothelial Progenitor Cells
Biomedicines, 11 (2023), 11; 3051. https://doi.org/10.3390/biomedicines11113051
Altabas, Velimir; Marinković Radošević, Jelena; Špoljarec, Lucija; Uremović, Stella; Bulum, Tomislav

Citirajte ovaj rad

Altabas, V., Marinković Radošević, J., Špoljarec, L., Uremović, S. i Bulum, T. (2023). The Impact of Modern Anti-Diabetic Treatment on Endothelial Progenitor Cells. Biomedicines, 11. (11). doi: 10.3390/biomedicines11113051

Altabas, Velimir, et al. "The Impact of Modern Anti-Diabetic Treatment on Endothelial Progenitor Cells." Biomedicines, vol. 11, br. 11, 2023. https://doi.org/10.3390/biomedicines11113051

Altabas, Velimir, Jelena Marinković Radošević, Lucija Špoljarec, Stella Uremović i Tomislav Bulum. "The Impact of Modern Anti-Diabetic Treatment on Endothelial Progenitor Cells." Biomedicines 11, br. 11 (2023). https://doi.org/10.3390/biomedicines11113051

Altabas, V., et al. (2023) 'The Impact of Modern Anti-Diabetic Treatment on Endothelial Progenitor Cells', Biomedicines, 11(11). doi: 10.3390/biomedicines11113051

Altabas V, Marinković Radošević J, Špoljarec L, Uremović S, Bulum T. The Impact of Modern Anti-Diabetic Treatment on Endothelial Progenitor Cells. Biomedicines [Internet]. 14.11.2023. [pristupljeno 09.11.2024.];11(11). doi: 10.3390/biomedicines11113051

V. Altabas, J. Marinković Radošević, L. Špoljarec, S. Uremović i T. Bulum, "The Impact of Modern Anti-Diabetic Treatment on Endothelial Progenitor Cells", Biomedicines, vol. 11, br. 11, Studeni 2023. [Online]. Dostupno na: https://urn.nsk.hr/urn:nbn:hr:105:665442. [Citirano: 09.11.2024.]

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Naslov (engleski)The Impact of Modern Anti-Diabetic Treatment on Endothelial Progenitor Cells
AutorVelimir Altabas
AutorJelena Marinković Radošević
AutorLucija Špoljarec
AutorStella Uremović
AutorTomislav Bulum
Autorova ustanovaSveučilište u Zagrebu
Medicinski fakultet
(KATEDRA ZA INTERNU MEDICINU)
Znanstveno / umjetničko
područje, polje i grana		BIOMEDICINA I ZDRAVSTVO
Kliničke medicinske znanosti
Interna medicina
Sažetak (engleski)
Diabetes is one of the leading chronic diseases globally with a significant impact on mortality. This condition is associated with chronic microvascular and macrovascular complications caused by vascular damage. Recently, endothelial progenitor cells (EPCs) raised interest due to their regenerative properties. EPCs are mononuclear cells that are derived from different tissues. Circulating EPCs contribute to regenerating the vessel’s intima and restoring vascular function. The ability of EPCs to repair vascular damage depends on their number and functionality. Diabetic patients have a decreased circulating EPC count and impaired EPC function. This may at least partially explain the increased risk of diabetic complications, including the increased cardiovascular risk in these patients. Recent studies have confirmed that many currently available drugs with proven cardiovascular benefits have beneficial effects on EPC count and function. Among these drugs are also medications used to treat different types of diabetes. This manuscript aims to critically review currently available evidence about the ways anti-diabetic treatment affects EPC biology and to provide a broader context considering cardiovascular complications. The therapies that will be discussed include lifestyle adjustments, metformin, sulphonylureas, gut glucosidase inhibitors, thiazolidinediones, dipeptidyl peptidase 4 inhibitors, glucagon-like peptide 1 receptor analogs, sodium-glucose transporter 2 inhibitors, and insulin.
Ključne riječi (engleski)
Jezikengleski
Vrsta publikacijeZnanstveni rad - Pregledni rad
Status objaveObjavljen
Vrsta recenzijeRecenziran
Verzija publikacijeObjavljena verzija rada (izdavačev PDF)
Naslov časopisaBiomedicines
Brojčani podacivol. 11, br. 11, 3051
e-ISSN2227-9059
DOIhttps://doi.org/10.3390/biomedicines11113051
URN:NBNurn:nbn:hr:105:665442
Datum objave publikacije2023-11-14
URL dokumentahttps://www.mdpi.com/2227-9059/11/11/3051
Vrsta resursaTekst
Prava pristupaOtvoreni pristup
Uvjeti korištenjaIkona licence Imenovanje 4.0 međunarodna (CC BY 4.0)
Datum i vrijeme pohrane2024-04-12 10:56:51